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The effects of patient sera following pomegranate consumption on proliferation and apoptosis induction on LNCaP prostate cancer cells in vitro were measured. Similar results were reported by Azadzoi et al. who also reported an increased intracavernosal blood flow, improvement of erectile response, and smooth muscle relaxation in an animal model. In an effort to search for markers of oxidative stress in arteriogenic ED and to investigate the protective role of dietary antioxidants, Zhang et al. developed atherosclerosis-induced ED in rabbits by balloon deendothelialization of the iliac arteries. Moreover, these studies also showed that pomegranate extracts not only inhibit the activation of NF-κB, but also delay the emergence of castration resistance in a xenograft model through reduced proliferation and increased apoptosis. The most striking evidence for the biological activity of pomegranate extracts was demonstrated in prostate cancer cells.
In both conditions, an increase of smooth muscle tension is thought to play a central role in the pathophysiology. The risk of prostate cancer may be reduced with the prophylactic intake of anti-inflammatory drugs. Evidence supports the role of proliferative inflammatory atrophy as a precursor to prostatic intraepithelial neoplasia and invasive prostate cancer . Pomegranate phytochemicals have been shown to be effective in reducing oxidative stress and to modulate inflammatory pathways 9, 10. Preclinical and clinical studies provide evidence of their antiproliferative properties and allyoutubes.com modulatory effects on inflammatory pathways . In order to explain preventive as well as therapeutic effects of nutrition components, investigators have focused on the antioxidative effects of plant polyphenols .
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